The Association Between Depressive Symptoms and N-Terminal Pro-B-Type Natriuretic Peptide With Functional Status in Patients With Heart Failure.

The Association Between Depressive Symptoms and N-Terminal Pro-B-Type Natriuretic Peptide With Functional Status in Patients With Heart Failure.

[Anonymous].  2018.  The Association Between Depressive Symptoms and N-Terminal Pro-B-Type Natriuretic Peptide With Functional Status in Patients With Heart Failure.. The Journal of cardiovascular nursing. 33(4):378-383.

A temperature dependent transition in the Pribnow box of the trp promoter.

Induction of altered hepatic foci in rats by the administration of hypolipidemic peroxisome proliferators alone or following a single dose of diethylnitrosamine.

Induction of altered hepatic foci in rats by the administration of hypolipidemic peroxisome proliferators alone or following a single dose of diethylnitrosamine.

[Anonymous].  1986.  Induction of altered hepatic foci in rats by the administration of hypolipidemic peroxisome proliferators alone or following a single dose of diethylnitrosamine.. Cancer research. 46(9):4601-6.

NMR studies of the trp repressor from Escherichia coli. Characterisation and assignments of residue types.

Validation of nigrostriatal positron emission tomography measures: critical limits.

Preliminary identification of the secondary structure of the trp repressor from Escherichia coli.

The phenotypic stability of altered hepatic foci: effects of withdrawal and subsequent readministration of phenobarbital.

Stress, cognitive appraisal, coping, and event free survival in patients with heart failure.

Identification of surface residues in the trp repressor of Escherichia coli.

A method to quantitate the relative initiating and promoting potencies of hepatocarcinogenic agents in their dose-response relationships to altered hepatic foci.

A method to quantitate the relative initiating and promoting potencies of hepatocarcinogenic agents in their dose-response relationships to altered hepatic foci.

[Anonymous].  1987.  A method to quantitate the relative initiating and promoting potencies of hepatocarcinogenic agents in their dose-response relationships to altered hepatic foci.. Carcinogenesis. 8(10):1491-9.