Structure-guided functional characterization of enediyne self-sacrifice resistance proteins, CalU16 and CalU19.

Achieving CNS axon regeneration by manipulating convergent neuro-immune signaling.

Derivation of multivariate syndromic outcome metrics for consistent testing across multiple models of cervical spinal cord injury in rats.

Subcellular localization and RNAs determine FUS architecture in different cellular compartments.

Self-assembled FUS binds active chromatin and regulates gene transcription.

Immune activation promotes depression 1 month after diffuse brain injury: a role for primed microglia.

IL-4 signaling drives a unique arginase+/IL-1β+ microglia phenotype and recruits macrophages to the inflammatory CNS: consequences of age-related deficits in IL-4Rα after traumatic spinal cord injury.

IL-4 signaling drives a unique arginase+/IL-1β+ microglia phenotype and recruits macrophages to the inflammatory CNS: consequences of age-related deficits in IL-4Rα after traumatic spinal cord injury.

[Anonymous].  2014.  IL-4 signaling drives a unique arginase+/IL-1β+ microglia phenotype and recruits macrophages to the inflammatory CNS: consequences of age-related deficits in IL-4Rα after traumatic spinal cord injury.. The Journal of neuroscience : the official journal of the Society for Neuroscience. 34(26):8904-17.

Manganese superoxide dismutase promotes interaction of actin, S100A4 and Talin, and enhances rat gastric tumor cell invasion.

Is neuroinflammation in the injured spinal cord different than in the brain? Examining intrinsic differences between the brain and spinal cord.

Is neuroinflammation in the injured spinal cord different than in the brain? Examining intrinsic differences between the brain and spinal cord.

[Anonymous].  2014.  Is neuroinflammation in the injured spinal cord different than in the brain? Examining intrinsic differences between the brain and spinal cord. Experimental neurology. 258:112-20.

Sulfiredoxin Promotes Colorectal Cancer Cell Invasion and Metastasis through a Novel Mechanism of Enhancing EGFR Signaling.