Focal cerebral ischemia and mitochondrial dysfunction in the TNFα-transgenic rat.

The actin cytoskeleton inhibits pore expansion during PIV5 fusion protein-promoted cell-cell fusion.

Directing dopaminergic fiber growth along a preformed molecular pathway from embryonic ventral mesencephalon transplants in the rat brain.

Directing dopaminergic fiber growth along a preformed molecular pathway from embryonic ventral mesencephalon transplants in the rat brain.

[Anonymous].  2011.  Directing dopaminergic fiber growth along a preformed molecular pathway from embryonic ventral mesencephalon transplants in the rat brain.. Journal of neuroscience research. 89(5):619-27.

Entry and fusion of emerging paramyxoviruses.

Targeting mitochondrial function for the treatment of acute spinal cord injury.

Mitochondria, Amyloid β, and Alzheimer's Disease.

Side chain packing below the fusion peptide strongly modulates triggering of the Hendra virus F protein.

Post-injury administration of the mitochondrial permeability transition pore inhibitor, NIM811, is neuroprotective and improves cognition after traumatic brain injury in rats.

Post-injury administration of the mitochondrial permeability transition pore inhibitor, NIM811, is neuroprotective and improves cognition after traumatic brain injury in rats.

[Anonymous].  2011.  Post-injury administration of the mitochondrial permeability transition pore inhibitor, NIM811, is neuroprotective and improves cognition after traumatic brain injury in rats.. Journal of neurotrauma. 28(9):1845-53.

C-terminal tyrosine residues modulate the fusion activity of the Hendra virus fusion protein.

Traumatic brain injury and trichloroethylene exposure interact and produce functional, histological, and mitochondrial deficits.