Immunological detection of glycated proteins in normal and streptozotocin-induced diabetic rats using anti hexitol-lysine IgG.

Fragmentation of ceruloplasmin following non-enzymatic glycation reaction.

An additional mitochondrial tRNA(Ile) point mutation (A-to-G at nucleotide 4295) causing hypertrophic cardiomyopathy.

Oxidative stress caused by glycation of Cu,Zn-superoxide dismutase and its effects on intracellular components.

Oxidative stress caused by glycation of Cu,Zn-superoxide dismutase and its effects on intracellular components.

[Anonymous].  1996.  Oxidative stress caused by glycation of Cu,Zn-superoxide dismutase and its effects on intracellular components.. Nephrology, dialysis, transplantation : official publication of the European Dialysis and Transplant Association - European Renal Association. 11 Suppl 5:34-40.

Glycated Cu,Zn-superoxide dismutase in rat lenses: evidence for the presence of fragmentation in vivo.

Leprosy in Myanmar, epidemiological and operational changes, 1958-92.

The expanding clinical phenotype of the tRNA(Leu(UUR)) A-->G mutation at np 3243 of mitochondrial DNA: diabetic embryopathy associated with mitochondrial cytopathy.

The expanding clinical phenotype of the tRNA(Leu(UUR)) A-->G mutation at np 3243 of mitochondrial DNA: diabetic embryopathy associated with mitochondrial cytopathy.

[Anonymous].  1996.  The expanding clinical phenotype of the tRNA(Leu(UUR)) A-->G mutation at np 3243 of mitochondrial DNA: diabetic embryopathy associated with mitochondrial cytopathy.. American journal of medical genetics. 62(4):404-9.

Glycation and inactivation of sorbitol dehydrogenase in normal and diabetic rats.

Reducing sugars trigger oxidative modification and apoptosis in pancreatic beta-cells by provoking oxidative stress through the glycation reaction.

Reducing sugars trigger oxidative modification and apoptosis in pancreatic beta-cells by provoking oxidative stress through the glycation reaction.

[Anonymous].  1996.  Reducing sugars trigger oxidative modification and apoptosis in pancreatic beta-cells by provoking oxidative stress through the glycation reaction.. The Biochemical journal. 320 ( Pt 3):855-63.

Clinical, physiological, and histological features in a kindred with the T3271C melas mutation.