Characterization of 3H estradiol-17 beta-(beta-D-glucuronide) binding sites in basolateral and canalicular liver plasma membranes.

Hepatic clearance and biliary secretory rate maximum of taurocholate in the recirculating and single pass isolated perfused rat liver. Effects of the cholestatic agent, estradiol-17 beta-(beta-D-glucuronide).

Hepatic clearance and biliary secretory rate maximum of taurocholate in the recirculating and single pass isolated perfused rat liver. Effects of the cholestatic agent, estradiol-17 beta-(beta-D-glucuronide).

[Anonymous].  1991.  Hepatic clearance and biliary secretory rate maximum of taurocholate in the recirculating and single pass isolated perfused rat liver. Effects of the cholestatic agent, estradiol-17 beta-(beta-D-glucuronide).. Biochemical pharmacology. 41(3):431-7.

Mirex inhibits bile acid secretory function in vivo and in the isolated perfused rat liver.

Canalicular transport: discovery of ATP-dependent mechanisms.

Carrier-mediated electrogenic transport of estradiol-17 beta-glucuronide in rat liver BMV.

Prolactin increases hepatic Na+/taurocholate co-transport activity and messenger RNA post partum.

Expression of an epidermal keratin protein in liver of transgenic mice causes structural and functional abnormalities.

Prolactin increases mRNA encoding Na(+)-TC cotransport polypeptide and hepatic Na(+)-TC cotransport.

MDR1 substrates/modulators protect against beta-estradiol-17beta-D-glucuronide cholestasis in rat liver.

ATP-dependent transport of beta-estradiol 17-(beta-D-glucuronide) in rat canalicular membrane vesicles.