Four early contributors to neurosurgery in North America.

Systemic complement depletion diminishes perihematomal brain edema in rats.

Urinary retention following routine neurosurgical spine procedures.

Activation of p44/42 mitogen activated protein kinases in thrombin-induced brain tolerance.

Intracerebral hemorrhage-induced neuronal death.

Inducible cyclooxygenase-2 expression after experimental intracerebral hemorrhage.

Induction of colligin may attenuate brain edema following intracerebral hemorrhage.

Thrombin preconditioning, heat shock proteins and thrombin-induced brain edema.

Attenuation of intracerebral hemorrhage and thrombin-induced brain edema by overexpression of interleukin-1 receptor antagonist.

Mechanisms of edema formation after intracerebral hemorrhage: effects of extravasated red blood cells on blood flow and blood-brain barrier integrity.

Mechanisms of edema formation after intracerebral hemorrhage: effects of extravasated red blood cells on blood flow and blood-brain barrier integrity.

[Anonymous].  2001.  Mechanisms of edema formation after intracerebral hemorrhage: effects of extravasated red blood cells on blood flow and blood-brain barrier integrity.. Stroke. 32(12):2932-8.