Brain region-specific, age-related, alterations in mitochondrial responses to elevated calcium.

Nicotine attenuates morphological deficits in a contusion model of spinal cord injury.

X-irradiation reduces lesion scarring at the contusion site of adult rat spinal cord.

Proteasome or calpain inhibition does not alter cellular tau levels in neuroblastoma cells or primary neurons.

alpha-Synuclein: a potent inducer of tau pathology.

Mitochondrial localization of mu-calpain.

Synaptic mitochondria are more susceptible to Ca2+overload than nonsynaptic mitochondria.

Artifactual dendritic beading in rat spinal cord induced by perfusion with cold saline and paraformaldehyde.

Sustained calpain inhibition improves locomotor function and tissue sparing following contusive spinal cord injury.

High cyclophilin D content of synaptic mitochondria results in increased vulnerability to permeability transition.

High cyclophilin D content of synaptic mitochondria results in increased vulnerability to permeability transition.

[Anonymous].  2007.  High cyclophilin D content of synaptic mitochondria results in increased vulnerability to permeability transition.. The Journal of neuroscience : the official journal of the Society for Neuroscience. 27(28):7469-75.