A brief elevation of serum amyloid A is sufficient to increase atherosclerosis.

Prevention of renal apoB retention is protective against diabetic nephropathy: role of TGF-β inhibition.

Serum amyloid A3 is pro-atherogenic.

Elevated circulating TGF-β is not the cause of increased atherosclerosis development in biglycan deficient mice.

Serum Amyloid A Is an Exchangeable Apolipoprotein.

Coordinated post-transcriptional regulation of Hsp70.3 gene expression by microRNA and alternative polyadenylation.

Myeloid-specific IκB kinase β deficiency decreases atherosclerosis in low-density lipoprotein receptor-deficient mice.

Pregnane X receptor mediates dyslipidemia induced by the HIV protease inhibitor amprenavir in mice.

Bisphenol A increases atherosclerosis in pregnane X receptor-humanized ApoE deficient mice.

IKKβ links vascular inflammation to obesity and atherosclerosis.