Angiotensin II increases vascular proteoglycan content preceding and contributing to atherosclerosis development.

Serum amyloid A, but not C-reactive protein, stimulates vascular proteoglycan synthesis in a pro-atherogenic manner.

Reversibility of renal injury with cholesterol lowering in hyperlipidemic diabetic mice.

Renal accumulation of biglycan and lipid retention accelerates diabetic nephropathy.

Serum amyloid A in atherosclerosis.

Decreased body fat, elevated plasma transforming growth factor-β levels, and impaired BMP4-like signaling in biglycan-deficient mice.

Decreased body fat, elevated plasma transforming growth factor-β levels, and impaired BMP4-like signaling in biglycan-deficient mice.

[Anonymous].  2013.  Decreased body fat, elevated plasma transforming growth factor-β levels, and impaired BMP4-like signaling in biglycan-deficient mice.. Connective tissue research. 54(1):5-13.

Prevention of TGFβ induction attenuates angII-stimulated vascular biglycan and atherosclerosis in Ldlr-/- mice.

Deficiency of endogenous acute phase serum amyloid A does not affect atherosclerotic lesions in apolipoprotein E-deficient mice.

Deficiency of endogenous acute phase serum amyloid A does not affect atherosclerotic lesions in apolipoprotein E-deficient mice.

[Anonymous].  2014.  Deficiency of endogenous acute phase serum amyloid A does not affect atherosclerotic lesions in apolipoprotein E-deficient mice.. Arteriosclerosis, thrombosis, and vascular biology. 34(2):255-61.

Increased atherosclerosis in mice with increased vascular biglycan content.

Biglycan deficiency: increased aortic aneurysm formation and lack of atheroprotection.